It just goes until it runs into a receptor and is taken in by a cell. From the second you first digest food, some will be taken on by fat cells. However, it could be similar to pouring water into a funnel. Your fat cells could be giving up energy as fast or faster than your adding to it. It could be also not giving up energy at all. The cells can realize what they need, or if they have enough. Through millennia of evolution, the human body has survived famines through storing fat.
This method has worked. You just change your activity patterns and eating patterns. You'll have to create a calorie deficit to lose the excess calories your body has stored.
Consuming excess calories means you're eating more than your body burns off in a day. Some energy expenditure comes from body processes, such as your heart beating, breathing and digestion. The remainder of the energy you expend daily is due to physical activities, such as walking, cleaning the house, climbing stairs, biking or gardening. If you eat more than you burn in a day, your body stores the excess calories to use later when calories are scarce.
Some excess calories you consume from carbohydrates are converted to and stored as glycogen, a complex carbohydrate, in your body. Your body stores glycogen primarily in your muscle and liver cells. For this reason, weight loss slows down over time. Counting your daily calorie intake is a common tactic if you're trying to lose weight.
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Health Conditions Discover Plan Connect. Calories are the energy in food. Thus, if we take in more calories than we expend or excrete, the excess has to be stored, which means that we get fatter and heavier. So far, so obvious. But this law tells us nothing about why we take in more calories than we expend, nor does it tell us why the excess gets stored as fat. Specifically, why do fat cells accumulate fat molecules to excess? This is a biological question, not a physics one.
Why are those fat molecules not metabolized instead to generate energy or heat? And why do fat cells take up excessive fat in some areas of the body but not others? Saying that they do so because excess calories are consumed is not a meaningful answer. Answering these questions leads to consideration of the role that hormones—insulin, in particular—play in stimulating fat accumulation in different cells.
Insulin is secreted in response to a type of carbohydrate called glucose. When the amount of glucose rises in the blood—as happens after eating a carbohydrate-rich meal—the pancreas secretes more insulin, which works to keep the blood glucose level from getting dangerously high. Insulin tells muscle, organ and even fat cells to take up the glucose and use it for fuel. It also tells fat cells to store fat—including fat from the meal—for later use.
As long as insulin levels remain high, fat cells retain fat, and the other cells preferentially burn glucose and not fat for energy. The main dietary sources of glucose are starches, grains and sugars. In the absence of carbohydrates, the liver will synthesize glucose from protein. The more easily digestible the carbohydrates, the greater and quicker the rise in blood glucose.
Fiber and fat in foods slow the process. Thus, a diet rich in refined grains and starches will prompt greater insulin secretion than a diet that is not. Sugars—such as sucrose and high-fructose corn syrup—may play a key role because they also contain significant amounts of a carbohydrate called fructose, which is metabolized mostly by liver cells.
The result, according to the hormone hypothesis, is an ever greater proportion of the day that insulin in the blood is elevated, causing fat to accumulate in fat cells rather than being used to fuel the body.
As little as 10 or 20 calories stored as excess fat each day can lead over decades to obesity. The hormone hypothesis suggests that the only way to prevent this downward spiral from happening, and to reverse it when it does, is to avoid the sugars and carbohydrates that work to raise insulin levels.
Then the body will naturally tap its store of fat to burn for fuel. The switch from carbohydrate burning to fat burning, so the logic goes, might occur even if the total number of calories consumed remains unchanged. Cells burn the fat because hormones are effectively telling them to do so; the body's energy expenditure increases as a result. To lose excess body fat, according to this view, carbohydrates must be restricted and replaced, ideally with fat, which does not stimulate insulin secretion.
This alternative hypothesis of obesity implies that the ongoing worldwide epidemics of obesity and type 2 diabetes which stems to great extent from insulin resistance are largely driven by the grains and sugars in our diets.
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